Nazim, M., Awad, M., Basiony, A. (2015). BIOLOGICAL CONTROL OF ROOT-ROT DISEASE ON TOMATO. Menoufia Journal of Plant Protection, 40(2), 359-372. doi: 10.21608/mjapam.2015.322227
M.N. Nazim; M.A. Awad; A.A. Basiony. "BIOLOGICAL CONTROL OF ROOT-ROT DISEASE ON TOMATO". Menoufia Journal of Plant Protection, 40, 2, 2015, 359-372. doi: 10.21608/mjapam.2015.322227
Nazim, M., Awad, M., Basiony, A. (2015). 'BIOLOGICAL CONTROL OF ROOT-ROT DISEASE ON TOMATO', Menoufia Journal of Plant Protection, 40(2), pp. 359-372. doi: 10.21608/mjapam.2015.322227
Nazim, M., Awad, M., Basiony, A. BIOLOGICAL CONTROL OF ROOT-ROT DISEASE ON TOMATO. Menoufia Journal of Plant Protection, 2015; 40(2): 359-372. doi: 10.21608/mjapam.2015.322227
1(1) Department of Agricultural Botany,Faculty of Agriculture, Minufiya University
2(2) Institute of Plant Pathology, Agriculture Research Center, Giza
Abstract
ABSTRACT: Tomato genotype Ps 550 was noticed as the highly susceptible genotype between the five tested tomato genotypes, while tomato genotype F1 743 was revealed as the most tolerant genotype against root rot disease infection. Trichoderma viride, Trichoderma harzianum, Bacillus subtilis, Pseudomonas spp.1 and Pseudomonas spp.2 were selected as biological control against the most pathogenic isolates, i.e. Fusarium solani, Rhizoctonia solani and Alternaria solani that were selected as the most aggressive three isolates that inoculated on both the above mentioned tolerant and susceptible tomato genotypes, i.e. F1 743 and Ps 550, respectively. The biological control agents were affected the virulence of Fusarium solani on root rot disease incidence. Trichoderma viride was recorded 0.0 % pre-emergence of tolerant tomato genotype F1 743. Fusarium solani on the susceptible tomato genotype (Ps 550) , Pseudomonas spp 1 and 2 were recorded the lowest pre-emergence damping-off ( 10 % ), while Pseudomonas spp 2 was recorded the least post-emergence damping-off (6%). Survival plants were recorded in the case of Pseudomonas spp 2 (84 %).All tested bioagents great affected the disease incidence both on the tolerant and/or susceptible tomato genotypes with variations between their responses against the Rhizoctonia solani and Alternaria solani isolates.